02-06-2017, 02:29 AM
Insertnamehere Wrote:Because pain, fever and inflammation are induced mostly by the same signaling pathways, namely prostaglandins and most antipyretics/antiinflammatories/pain relievers target the same enzyme that produces them, COX-2.
I suspected something like that. But that common pathway is not identical up to the end. It is possible to target different parts of it resulting in a differential modulation of its various components. Someone should work on that, that was my point.
Quote:That said, there are pain-specific pharmaceuticals. These mess with the receptors of pain signals at the neuron level. Morphine and the likes will do this.
I will never mess with my pain receptors. My pain sensation is excellent (i.e. I have a very high pain tolerance), I don't want to fuck that up.
Lidocaine is a local anesthetic, which wouldn't be suitable to alleviate a headache.
Quote:Pain is a good thing too, you know? Without it you'd never know when something is going wrong with you and you wouldn't have made it pass childhood.
Yeah well, I wasn't asking for my whole ability to sense pain to be abolished. I'm one of the last people on this planet to argue against pain like that.
I had a fever, my airways were clogged up and I couldn't breathe half the time, I had a painful cough and a sore throat, my ears were shut (I don't know what's the term for it in English; it's the same thing that happens when you're on an airplane), and on top of that I had a headache for the second day in a row. It served no beneficial purpose and I would have liked to take away that one thing I could have controlled. As it stood, I didn't do it, because I chose not to lower my fever even though it would have meant getting rid of my headache.
''Do I look civilized to you?''